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donna j lang[Author]
(73 results)?
Circulating tumor DNA: current challenges for clinical utility.
J Clin Invest. 2022 Jun 15;132(12):e154941. doi: 10.1172/JCI154941.
J Clin Invest. 2022.
PMID: 35703177
Free PMC article.
Review.
Secretory pathway Ca2+-ATPase SPCA2 regulates mitochondrial respiration and DNA damage response through store-independent calcium entry.
Makena MR, Ko M, Mekile AX, Senoo N, Dang DK, Warrington J, Buckhaults P, Talbot CC Jr, Claypool SM, Rao R.
Makena MR, et al. Among authors: dang dk.
Redox Biol. 2022 Apr;50:102240. doi: 10.1016/j.redox.2022.102240. Epub 2022 Jan 17.
Redox Biol. 2022.
PMID: 35063802
Free PMC article.
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An integrated functional and clinical genomics approach reveals genes driving aggressive metastatic prostate cancer.
Das R, Sjöström M, Shrestha R, Yogodzinski C, Egusa EA, Chesner LN, Chen WS, Chou J, Dang DK, Swinderman JT, Ge A, Hua JT, Kabir S, Quigley DA, Small EJ, Ashworth A, Feng FY, Gilbert LA.
Das R, et al. Among authors: dang dk.
Nat Commun. 2021 Jul 29;12(1):4601. doi: 10.1038/s41467-021-24919-7.
Nat Commun. 2021.
PMID: 34326322
Free PMC article.
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Epigenetic Modulation of SPCA2 Reverses Epithelial to Mesenchymal Transition in Breast Cancer Cells.
Makena MR, Ko M, Dang DK, Rao R.
Makena MR, et al.
Cancers (Basel). 2021 Jan 12;13(2):259. doi: 10.3390/cancers13020259.
Cancers (Basel). 2021.
PMID: 33445642
Free PMC article.
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A Ca2+-ATPase Regulates E-cadherin Biogenesis and Epithelial-Mesenchymal Transition in Breast Cancer Cells.
Dang DK, Makena MR, Llongueras JP, Prasad H, Ko M, Bandral M, Rao R.
Dang DK, et al.
Mol Cancer Res. 2019 Aug;17(8):1735-1747. doi: 10.1158/1541-7786.MCR-19-0070. Epub 2019 May 10.
Mol Cancer Res. 2019.
PMID: 31076498
Free PMC article.
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NHA2 promotes cyst development in an in vitro model of polycystic kidney disease.
Prasad H, Dang DK, Kondapalli KC, Natarajan N, Cebotaru V, Rao R.
Prasad H, et al. Among authors: dang dk.
J Physiol. 2019 Jan;597(2):499-519. doi: 10.1113/JP276796. Epub 2018 Oct 17.
J Physiol. 2019.
PMID: 30242840
Free PMC article.
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